Changes in the composition of fat molecules on the membranes of nerve cells appear to change how the alpha-synuclein protein interacts with these same cell membranes, possibly promoting the aggregation that characterizes Parkinson’s disease, new research reports. The study, “The docking of synaptic vesicles on the presynaptic membrane induced by α-synuclein is modulated by lipid composition,” was published in the journal Nature Communications. Alpha-synuclein is a protein particularly abundant in the brain. When it misfolds and accumulates,…

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